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Potential effects of sCD40L, CD36, IL-23 and arginase-1 molecules on the pathogenesis of brucellosis

dc.contributor.authorKızmaz, Muhammed Ali
dc.contributor.authorEllergezen, Pınar Hız
dc.contributor.authorDemir, Nesrin
dc.contributor.authorCagan, Eren
dc.contributor.authorÇolak, Zehranur
dc.contributor.authorAkalın, Emin Halis
dc.contributor.authorOral, Haluk Barbaros
dc.contributor.authorBudak, Ferah
dc.date.accessioned2023-03-14T11:26:43Z
dc.date.available2023-03-14T11:26:43Z
dc.date.issued2021en_US
dc.identifier.issn0014-2980
dc.identifier.issn1521-4141
dc.identifier.urihttps://hdl.handle.net/20.500.12428/3806
dc.description.abstractBrucellosis is a systemic infectious disease that can be transmitted from animals to humans, ranging from mild to severe clinical pictures. In our study, we aimed to reveal the roles of sCD40L, CD36, IL‐23 and arginase‐1 (ARG1) molecules in the pathogenesis of brucellosis. sCD40L binds and activates CD40 on antigen‐presenting cells, thereby promoting the secretion of pro‐inflammatory cytokines and nitric oxide (NO) synthesis.en_US
dc.language.isoengen_US
dc.publisherWileyen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectBacterial infectionsen_US
dc.subjectEffector moleculesen_US
dc.subjectImmune response tracingen_US
dc.subjectInfectious diseaseen_US
dc.titlePotential effects of sCD40L, CD36, IL-23 and arginase-1 molecules on the pathogenesis of brucellosisen_US
dc.typeconferenceObjecten_US
dc.authorid0000-0003-3658-0185en_US
dc.relation.ispartofEuropean Journal of Immunologyen_US
dc.departmentFakülteler, Tıp Fakültesi, Temel Tıp Bilimleri Bölümüen_US
dc.identifier.volume51en_US
dc.identifier.issueSupp1en_US
dc.identifier.startpage319en_US
dc.identifier.endpage319en_US
dc.institutionauthorDemir, Nesrin
dc.relation.publicationcategoryKonferans Öğesi - Uluslararası - Kurum Öğretim Elemanıen_US
dc.authorwosidCLW-1910-2022en_US
dc.authorscopusid57218293761en_US
dc.identifier.wosqualityQ2en_US
dc.identifier.wosWOS:000753366401509en_US


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